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As in women, estrogen stimulates the growth of breast tissue in males. In addition to straight promoting male breast tissue growth, estrogens indirectly decrease secretion of testosterone by suppressing luteinizing hormone secretion, resulting in reduced testicular secretion of testosterone. In  تثدي الرجال الاسباب والعلاج , estrogens can increase blood levels of the protein sex hormone-binding globulin (SHBG), which binds free testosterone (the active form) resulting in decreased action of testosterone in male breast tissue.

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Klinefelter syndrome is a notable example of a condition that causes hypogonadism and gynecomastia, and has a greater danger of breast cancer in males (2050 times greater than males without the condition). Central hypogonadism (indicating an issue with the brain) leads to reduced production and release of luteinizing hormonal agent (LH, a stimulatory signal for endogenous steroid hormone synthesis) which causes decreased production of testosterone and estradiol in the testes.

Cirrhotics tend to have increased secretion of the androgenic hormonal agent androstenedione from the adrenal glands, increased conversion of this hormonal agent into different kinds of estrogen, and increased levels of SHBG, which results in reduced blood levels of totally free testosterone. Around 1040% of people with Tomb' disease (a common kind of hyperthyroidism) experience gynecomastia.

تثدي الرجل الكاذب

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Proper treatment of the hyperthyroidism can cause the resolution of the gynecomastia. Medications are understood to trigger gynecomastia through a number of various systems. These systems consist of increasing estrogen levels, imitating estrogen, reducing levels of testosterone or other androgens, obstructing androgen receptors, increasing prolactin levels, or through unknown ways. High levels of prolactin in the blood (which may take place as an outcome of particular tumors or as a side impact of specific medications) has actually been connected with gynecomastia.

التثدي عند الرجال

Receptors for prolactin and other hormones consisting of insulin-like development factor 1, insulin-like growth aspect 2, luteinizing hormone, progesterone, and human chorionic gonadotropin have actually been discovered in male breast tissue, however the impact of these numerous hormones on gynecomastia development is not well comprehended. Medical diagnosis [edit] To diagnose gynecomastia, an extensive history and physical exam are obtained by a doctor.